Whiteley M, Greenberg EP: Promoter specificity elements in Pseudo

Whiteley M, Greenberg EP: Promoter specificity elements in Pseudomonas aeruginosa quorum-sensing-controlled genes. J Bacteriol 2001,183(19):5529–5534. 10.1128/JB.183.19.5529-5534.20019544311544214CrossRefPubMedCentralPubMed 30. Schuster

M, Urbanowski ML, Greenberg EP: Promoter specificity in Pseudomonas aeruginosa quorum sensing revealed by DNA binding of purified LasR. Proc Natl Acad Sci U S A 2004,101(45):15833–15839. 10.1073/pnas.040722910152874115505212CrossRefPubMedCentralPubMed 31. Holloway BW, Krishnapillai V, Morgan AF: Chromosomal genetics of Pseudomonas . Microbiol learn more Rev 1979,43(1):73–102. 281463111024CrossRefPubMedCentralPubMed 32. Kovach ME, Elzer PH, Hill DS, Robertson GT, Farris MA, Roop RM 2nd, Peterson KM: Four new derivatives of the broad-host-range cloning vector pBBR1MCS, carrying different antibiotic-resistance cassettes. Gene 1995,166(1):175–176. PLX3397 purchase 10.1016/0378-1119(95)00584-18529885CrossRefPubMed

33. Marx CJ, Lidstrom ME: Broad-host-range cre-lox system for antibiotic marker recycling in gram-negative bacteria. Biotechniques 2002,33(5):1062–1067. 12449384CrossRefPubMed 34. Bouffartigues E, selleck Gicquel G, Bazire A, Bains M, Maillot O, Vieillard J, Feuilloley MG, Orange N, Hancock RE, Dufour A, Chevalier S: Transcription of the oprF gene of Pseudomonas aeruginosa is dependent mainly on the SigX sigma factor and is sucrose induced. J Bacteriol 2012,194(16):4301–4311. 10.1128/JB.00509-12341626422685281CrossRefPubMedCentralPubMed 35. Corbella ME, Puyet A: Real-time reverse transcription-PCR analysis of expression of halobenzoate and salicylate catabolism-associated operons in two strains of Pseudomonas aeruginosa . Appl Environ Microbiol 2003,69(4):2269–2275. 10.1128/AEM.69.4.2269-2275.200315480912676709CrossRefPubMedCentral 36. Smith AW, Iglewski BH: Transformation of Pseudomonas aeruginosa by electroporation. Nucleic Acids Res 1989,17(24):10509. 10.1093/nar/17.24.105093353342513561CrossRefPubMedCentralPubMed Competing interests The authors declare that they have no competing interests. Authors’ contributions

AB performed all the experiments and co-drafted the manuscript. AD supervised the study and co-drafted the manuscript. Both authors read and approved the final manuscript.”
“Background In order to generate effective mechanisms for the 4��8C control of plant diseases, it is crucial to gain insights into the diversity and population dynamics of plant pathogens [1, 2]. Pathogens showing a high genotypic diversity are regarded as being harder to control, because plant resistance can be overcome by more suitable pathotypes [3]. Hence, the development of durable resistance becomes more challenging with this kind of pathogens. Factors such as the genetic flow between pathogen populations and processes that increase the genetic changes of these populations may contribute to break the resistance in monocultures [3–5]. Xanthomonas axonopodis pv.

Comments are closed.